| Name |
Views and Comments | Date |
| dpmsurf |
The latest research is starting to point to the fact that this problem is huge. Chronic Inflammatory problems were named as a major cause of many of the BIG American diseases so prevalent today. It really seems that this is a very important aspect to increasing your health. The mainstream will not catch on to this for a while. Too much money involved.
The main issue here is that you can treat many problems by decreasing inflammation. The following can be linked to inflammation.
Heart disease
Alzheimer's
Colon cancer
Osteoarthritis
Rheumatoid Arthritis
High Blood Pressue
Muscle recovery
Increasing the ratio of Omega 3 to Omega 6 is an anti-inflammatory. some of the health benefits of Omega 3 related to inflammation. It can do the following
Suppressing T-cell activation and proliferation
Maintaining T-cell homeostasis through the contraction of populations of activated T cells
Reduction of inflammation cytokines and eicosanoids
Promotion of apoptosis
The above are all the scientific reasons for decreased inflammation due to Omega-3.
Omega 3 can help alleviate inflammation. Unlike drugs created for inflammation. Omega3 is systemic. It is not pointed at one thing – it helps all inflammation. This is an important point. Usually when you are sick only one problem manifests itself. Most likely there are other problems underneath the surface. SO by decreasing all Inflammation you may very well be really increasing your health. Instead of a band aid for what is ailing you at the time. |
6/18/2008 |
| Harry Horton |
Silent inflammation and its adverse effects most likely can be founded in the various modes of function of the mitochondrial electron transport chain. And furthermore, how the modes of function of the electron transport chain can be tied into the other constituent energy producing areas of the mitochondria (Kreb's Cycle and B oxidation), and even further extended back to cytoplasmic glycolysis. Normal regular healthy oxidative phosphorylation, within its process, the working in tandem of ATP hydrolysis along with ATP resynthesis, in a healthy timely sequential operation, is basic to normal health of the body. But sometimes, accidents happen. Say severe burns inflict the body from an industrial accident. Then the healthy normal status quo modes of function in the cell's mitochondrial electron transport chain disappear and are replaced by other modes of functions, that involve the predominance of uncoupling phenomena of the electron transport chain resulting in altered ATP production. This uncoupling mode accompanies inflammation. And the electron transport chain gives off heat as it dissipates hydrogen protons through leakage in the chain. But most interestingly, insulin resistance, the physiological condition heralding diabetes, also is embedded in constant inflammation. Shoelson of Joslin Diabetes Center has done much work in the first part of this decade on that phenomena.
Hence, the very interesting supposition. The emerging insulin resistance that appears, giving away to an eventual condition of diabetes, is really a switching mechanism gone bad in the body. Instead of healthy normal oxidative phosphorylation with consistent ATP synthesis apparent, rather in the prediabetic phase, low grade inflammation appears on a constant basis, with insulin resistance building within it. When the body's cells become unable to use insulin, in my belief, that condition is like a switching mechanism gone awry. A mechanism gone awry in a metamorphosizing manner, gradual and gradated in its stealthy oncoming, intensifying the developing diseased diabetes condition; such onsetting insulin resistance is within its bed of silent low grade inflammation that stays on as a fairly steady bodily occurence. Thus insulin resistance is a normal process in the body that becomes diseased with its dysfunctional nature when inflammation gains ascendency, especially in old age. The doctor Richard Fiddian Green, wrote an article in the British Medical Journal called "A Call to worship a new god, tissue pH." 28 June 2004. In the article he gives a good illustrative account in burn victims how inflammation occuring in the healing wound is reflective of the mitochondrial electron transport chain's 'switching modes of function' and how it occurs in such burn victims. Ingenously using room temperature, and elevating it, Fiddian Green utilized this fact in his study. A quote from the article goes as follows: "Lionel Opie claims the generation of heat in myocytes is determined by the magnitude of the proton leak rate across the mitochondrial membranes If so it is a function of both the protonmotive force and the permeability of the mitochondrial membrane or degree of openess of the permeability transition pore. Support for this claim is provided by the marked reduction in oxygen consumption that occurs when an occlusive dressing and elevation in room temperature is used to reduce heat loss from a burn wound, healing wounds generating their ATP needs by anaerobic glycolysis rather than by oxidative phosphorylation (6). The observation further suggests that oxidative phosphorylation might be largely uncoupled in a healing wound." End of quote. Severe burn wounds in such the above scenario are accompanied by a roaring rapidly operable inflammation state indigenous to the body's healing processes. And the above R.F. Green quote nicely displays the switching mechanism of the mitochondrial electron chain 'mode of function" from relative normal healthy oxidative phosphorylation to one of the chain developing an uncoupling characteristic, in order to address the extraordinary condition of bodily burn wounds. Note how elevated room temperature acts as a compensatory aid to keeping the body warm. The body's cells are giving off heat in the uncoupling mode, and the threat of heat loss that impedes therapeutic progress of the body gowing cold, (like impending death situations, to some degree) is actual in such the above scenario. Finally the uncoupling phenomena and the inflammatory state it resides in are well within the accurate switching mechanism mode of the electron transport chain processes. In diabetes development, this clean, concise, in timely manner switching mechanism mode, disappears and is replaced by an unwieldly breakdown of the synchronization of modes of function of the electron transport chain. Yet the same componenets of inflamation, insulin resistance, and uncoupling protein predominance in the electron transport chain exist in the emergent diabetic case as well as in the previously illustrated burn wound victim case. The above theoretical assertions seem to be rather valid, and future research hopefully will further elucidate with more exactness how inflammation creates deleterious diseased conditions such as diabetes. in the meantime the above commentary still must be considered in a theoretical and conjectural light. But its interesting.
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6/2/2007 |
| angie |
How about not putting your self in danger? Just don't take it!!!!! |
7/20/2006 | |
