Atkins Diet Risks Detailed in Medical Translation Report

For the last ten years, the Atkins diet has received a great deal of publicity, praise and criticism. The diet, developed by Dr Robert Atkins takes a controversial approach to weight loss by encouraging a high protein and fat diet instead of a low-carbohydrate diet. The diet has been marketed through a series of books, weightless programs, licensed-proprietary branding for food products and related items. But ever since the diet’s release, heart specialists and public health experts have criticized the diet out of fear that it would cause heart disease.

According to a medical research report that was published in Mexico and later disseminated by a Medical Translation firm, a recent study confirmed that the diet maybe dangerous. The findings from the study suggested that the Atkins diet could cause ketosis. It’s important to realize that ketosis is merely means that our bodies are using fat for energy. When fat is broken down for energy, most of it gets converted to ATP (energy) and the remainder is converted to ketones for energy.

Some vital organs prefer ketones to glucose and most cells in the brain are able to use ketones for part of their energy. But there is one type of ketone molecule, called acetone, that cannot be used and is excreted as waste, mostly in the urine and breath. However, the process of ketosis produces methylgloxal - which can damage blood vessel and tissue damage.

In the report, the translation professional discusses a 53-year old man showed who the possible risks associated with the diet. Before going on the diet, the research subject had low cholesterol and LDL levels and no evidence of coronary disease. After three years of being on the Atkins diet, his cholesterol and LDL rose to high risk levels and he developed blood circulation and chest pains. After being hospitalized for chest pains and severe narrowing of a coronary artery, he went off the Atkins diet and his health improved to pre-Atkins levels.

By Sarah Hudson
Published: 11/16/2009
 
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